By Peter M. Haddad, Serdar Dursun, Bill Deakin
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2003). The triad comprises: ◆ Altered mental status; agitation, excitement and confusion ◆ Neuromuscular hyperactivity; tremor, clonus, myoclonus, hyperreflexia, and (in the advanced stage) pyramidal rigidity ◆ Autonomic hyperactivity; diaphoresis, fever, mydriasis, tachycardia and tachypnoea. The onset of frank toxicity is usually rapid because it results from drug combinations and starts when the second drug is introduced.
3), 115–21. Osser DN (1999). Neuroleptic induced parkinsonism. ) Movement Disorders in Neurology and Neuropsychiatry, 2nd edn, pp. 61–8. Blackwell Science: Malden, MA. Summary: Tardive dyskinesia Definition Various involuntary movements (including myoclonic jerks, tics, chorea and dystonia but not tremor) caused by a psychotropic drug and appearing late in the course of treatment. Incidence Cumulative incidence of about 5% per years in patients starting conventional antipsychotic treatment (Kane et al.
The neuroleptic malignant syndrome; agent and host interaction. Acta Psychiatr Scand, 73, 337–47. Shalev A, Hermesh H, and Munitz H (1989). Mortality from neuroleptic malignant syndrome. J Clin Psychiatry, 50, 18–25. Stauder KH (1934). Die Toldliche katatinie. Arch Psychiatr Nervenkranke, 102, 614–34. Susman VL and Addonizio G (1988). Recurrence of neuroleptic malignant syndrome. J Nerv Ment Dis, 176, 234–41. White DA and Robins AH (1991). Catatonia: harbinger of the neuroleptic malignant syndrome.
Adverse syndromes and psychiatric drugs: a clinical guide by Peter M. Haddad, Serdar Dursun, Bill Deakin