Get ABC of Clinical Haematology, 3rd Edition (ABC Series) PDF

By Drew Provan

ISBN-10: 1405153539

ISBN-13: 9781405153539

ISBN-10: 1444312685

ISBN-13: 9781444312683

This ABC has validated itself as a favored advent to scientific Haematology, correct either for postgraduate scholars and first care practitioners. Now in its 3rd version, the ABC of medical Haematology has been completely revised, that includes the newest remedies for leukemia, antithrombotics and medicine for lymphoma and masking the newest advances in hematology and bone marrow transplantation. With every one bankruptcy written by way of experts of their respective fields, this can be a fantastic simple textual content for normal perform and health center employees with sufferers who've blood comparable difficulties, specifically GPs, hematology nurses, junior medical professionals, hematologists, and trainees in hematology.

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Extra resources for ABC of Clinical Haematology, 3rd Edition (ABC Series)

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The Myeloproliferative Disorders. Blackwell Publishing, Oxford, 2006. 7 Leucoerythroblastic blood film in a patient with idiopathic myelofibrosis. Note the nucleated red blood cell (arrowhead) and the myelocyte (arrow). We thank Dr Ellie Nacheva for the fluorescence in situ hybridization image showing deletion of the long arm of chromosome 20. g. imatinib) the duration of chronic phase disease is much longer than with earlier therapy • TKIs are therefore the best initial therapy for CML • Allografting is currently reserved for patients who fail TKIs Chronic myeloid leukaemia (CML) is a clonal malignant myeloproliferative disorder believed to originate in a single abnormal haemopoietic stem cell.

Druker BJ, Guilhot P, O’Brien SG et al. Five-year follow-up of imatinib therapy for newly diagnosed chronic-phase chronic myeloid leukemia. New Eng J Med 2006; 355: 2408–17. Goldman JM, Druker B. Chronic myeloid leukemia: current treatment options. Blood 2001; 98: 2039–42. Goldman JM, Melo JV. Chronic myeloid leukemia. New England Journal of Medicine 2003; 349: 1449–62. Hughes T, Deininger M, Hochhaus A et al. Monitoring CML patients responding to treatment with tyrosine kinase inhibitors – recommendations for ‘harmonizing’ current methodology for detecting BCR-ABL transcripts and kinase domain mutations and for expressing results.

6 Spontaneous skin purpura in severe autoimmune thrombocytopenia. 7 Bone marrow aspirate showing increased megakarocytes in immune thrombocytopenia. 2 Disorders with increased consumption of platelets • Disorders with immune mechanism • Autoimmune: idiopathic thrombocytopenic purpura • Alloimmune: post-transfusion purpura, neonatal alloimmune thrombocytopenia • Infection-associated: infectious mononucleosis, HIV, malaria • Drug-induced: heparin, penicillin, quinine, sulphonamides, rifampicin • Thrombotic thrombocytopenic purpura/haemolytic uraemic syndrome • Hypersplenism and splenomegaly • Disseminated intravascular coagulation • Massive transfusion Post-transfusion purpura Post-transfuson purpura is a rare complication of blood transfusion.

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ABC of Clinical Haematology, 3rd Edition (ABC Series) by Drew Provan


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